Researchers at Northwestern Medicine have made significant strides in understanding heart failure with preserved ejection fraction (HFpEF), a life-threatening condition with few treatment options. The study highlights how chronic diseases contribute to HFpEF by damaging the heart and other systems, emphasizing inflammation's central role. By utilizing advanced techniques to analyze immune cells in HFpEF models, scientists discovered elevated inflammatory markers linked to the disease. These insights pave the way for potential therapies targeting inflammation, giving hope for better management of HFpEF.
"We've known for years that inflammation plays an important role in this kind of heart failure, but the mechanisms and what exactly was happening was not well understood."
"The findings are a good reminder that the heart exists within this whole body that's experiencing these systemic comorbidities."
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