Our findings reveal a bidirectional relationship between sleep and cardiovascular health, suggesting that sleep mediates processes crucial for heart healing and managing inflammation.
In both human and mouse models, myocardial infarction prompts the recruitment of monocytes to the brain, indicating a significant connection between cardiac events and sleep regulation.
The recruited monocytes play a vital role in enhancing sleep following myocardial infarction, which in turn helps in regulating sympathetic nervous system activity.
This study underscores the importance of understanding the interplay between sleep and cardiovascular pathology to better address issues related to myocardial infarction and recovery.
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