Neuroinflammation triggers a cascade of immune changes in the brain that affects muscles, cause fatigue, and offer potential treatment avenues.
By studying neuroinflammation across diseases like COVID-19 and Alzheimer's, we uncovered how cytokines alter muscle performance without damaging fibers.
Our findings show that cytokines released during neuroinflammatory responses disrupt mitochondrial function in muscle cells, leading to fatigue rather than damage.
Identifying the connection between brain inflammation and muscle fatigue can lead to innovative therapies targeting neuroinflammatory pathways.
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