Lithium deficiency has emerged as a potential cause of Alzheimer's disease, prompting researchers from Harvard University to investigate its role in brain function. They administered lithium orotate to mice, effectively preventing memory loss and the associated pathological changes related to dementia. The research team emphasizes caution against self-supplementation and advocates for clinical trials involving humans. Their findings are documented in the journal Nature, adding a significant perspective to understanding Alzheimer's, which has long been obscured by complexity and confusion regarding its origins and progression.
Research leader Bruce A. Yankner compares Alzheimer's to a chaotic battlefield covered in corpses after a war. The destruction makes it hard to know how it all began.
Lithium deficiency is the breakdown in communication that leads to war, according to Bruce A. Yankner, providing a new theoretical framework for understanding Alzheimer's.
The Harvard team aimed to explore the role of around 30 metallic chemical elements, such as iron and copper, in the development of Alzheimer's disease.
Administering lithium orotate to mice prevented memory loss and the pathological changes associated with dementia, establishing a potential treatment avenue.
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